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Break Out The Fire Hose! *Inflammation and Autoimmune Diseases

With the discovery of pathogenic bacteria and the advent of proper hygiene, death from these problems became less common.

FOR IMMEDIATE RELEASE

PRLog (Press Release) - Jun 20, 2009 -
Inflammation has become the new epicenter of disease, disability and death.*We are a people on fire!

*Inflammation and the immune system

*Our immune system uses heat to destroy invading bacteria. A specialized cell called a macrophage douses the invader with inflammatory enzymes (like acids) and the heat generated in this battle destroys the invading bacteria.

*Sometimes our own healthy cells become the focus for disease. This often happens because of toxic exposure. When we eat, drink or breathe a toxin, or if it is absorbed through our skin, our detoxification system works to remove it before the poison can compromise our health. Unfortunately, many of today’s poisons are simply too strong and they overwhelm our detoxification system and enter healthy cells. These cells now become the target of disease, infection and inflammation. Our immune system must treat them as hostile and use heat to destroy them.

*Autoimmune diseases arise when our immune system becomes so overworked that it can no longer tell the difference between healthy cells and sick cells. It attacks all cells of a particular type equally. This body system is never able to heal and chronic inflammation sets in.

*The weakest link

*A chain is only as strong as its weakest link. You are only as healthy as your weakest body system. How robust you are depends mostly on lifestyle choices, somewhat on your environment and a little on your genetics.

*When a toxin overwhelms your detoxification process it will likely settle in your weakest system. The immune system melts these polluted cells in an effort to save healthy cells. This is acute inflammation. With chronic toxicity comes the opportunity for chronic inflammation. In the case of chronic inflammation, the immune system attacks any sick cell as well as any healthy cell that looks like the sick one. If the immune system attacks connective tissue we say a person has lupus. If it attacks the beta cells of the pancreas we say they have type 1 diabetes. If it attacks the joints we say they have rheumatoid arthritis.

*These are only a few of the more than 200 autoimmune diseases. Yet they all share one common attribute: inflammation.

*Putting the fire out

*Inflammation arises because of:

* Toxins
* Injuries
* Emotional distress
* Nutrient deficiency

*We simply must reduce our toxic exposure to reduce the chances of developing an autoimmune disorder. And we must also develop emotional coping strategies and have all of the nutrients we need in our body in the right quantity – every moment of every day. These are the first steps to reducing inflammation.

*We can directly douse the fire with anti-inflammatory Betalains such as those found in TriVita's Nopalea. A Betalain is a protein-antioxidant complex found in certain plants grown in hot climates. The hotter the climate, the more Betalains the plant needs to survive and the more we can benefit.

*Information above provided by Brazos Minshew, TriVita's Chief Science Officer
TriVita's Weekly Wellness Report June 20th. 2009

To learn more about Betalains and Nopalea success stories visit here now >http://sonoranbloom.com/13115780

The above information presented herein is intended for educational purposes only. These statements have not been evaluated by the FDA and are not intended to diagnose, cure, treat or prevent disease. Individual results may vary, and before using any supplements, it is always advisable to consult with your own health care provider.

# # #

Trivita a debt Free nutritional company with a 9 year track record and over $100,000,000 in sales is setting the pace for the wellness industry.

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Contact Email:
***@gmail.com
Source:Jack Shea
Zip:99362
City/Town:Walla Walla
State/Province:Washington
Country:United States
Industry:Health, Medical, Business
Tags:, , , , ,
Last Updated:Jun 20, 2009
Shortcut:http://prlog.org/10263056
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